MECHANISM: Decrease in arteriolar resistance via vasodilation. This arterial vasodilation goes unmatched by the venous circulation, which CCB do not effect. This is a disproportionate change in resistance increasing hydrostatic pressure in the capillary circulation and causing fluid to shift into the interstitium. Hence, edema!
REFERENCES
Domenic A. Sica. Calcium channel blocker-related periperal edema: can it be resolved? J Clin Hypertens (Greenwich) 2003 Jul-Aug; 5(4): 291-4, 297.
Older age is recognized as a risk factor for obstructive sleep apnea.[1] But why? Suspected…
Being overweight (BMI ≥ 25 kg/m2)/Obese (BMI ≥ 30 kg/m2) is one of the most…
Nocturia is a common symptom associated with obstructive sleep apnea. [1] Img Cred: Am J…
ASD (Atrial Septal Defect) Wide, Fixed split S2 (in contrast to the normal variation in…
Mixed apneas are characterized by absent respiratory effort and airflow in the first section of…
Although rare, the differential diagnosis of hypoventilation and hypercapnia respiratory failure includes hypothyroidism. It is…
This website uses cookies.